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Oxidative stress in endometriosis activates key proliferation pathways including Raf/MEK/ERK and mTOR, promotes extracellular matrix degradation via matrix metalloproteinases, drives angiogenesis through VEGF upregulation, and sustains neurogenic inflammation through macrophage activation and prostaglandin synthesis

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However, the optimal timing depends on the specific plant compound and product formulation

Glutathione is so important that cell death results when levels inside cells drop too low